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Image Unavailable-19399-2.5L

Nitric acid : Dipicolinic acid (Konzentrat) (17 mM : 17 mM) in Wasser Eluenskonzentrat für IC (10x) for Metrosep C 6, Supelco®

Lieferant: Merck
Beschreibung: Nitric acid : Dipicolinic acid (Konzentrat) (17 mM : 17 mM) in Wasser Eluenskonzentrat für IC (10x) for Metrosep C 6, Supelco®

Zertifikate

Image Unavailable-61905-2.5L

Nitric acid : Dipicolinic acid (34 mM : 14 mM) in Wasser Eluenskonzentrat für IC (20x) for Metrosep C 4, Supelco®

Lieferant: Merck
Beschreibung: Nitric acid : Dipicolinic acid (34 mM : 14 mM) in Wasser Eluenskonzentrat für IC (20x) for Metrosep C 4, Supelco®

Zertifikate

Image Unavailable-BOSSBS-11997R-A555

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 555)

Artikel-Nr: (BOSSBS-11997R-A555)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-A488

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 488)

Artikel-Nr: (BOSSBS-11997R-A488)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-FITC

Anti-DDAH1 Rabbit Polyclonal Antibody (FITC (Fluorescein Isothiocyanate))

Artikel-Nr: (BOSSBS-11997R-FITC)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-CY3

Anti-DDAH1 Rabbit Polyclonal Antibody (Cy3®)

Artikel-Nr: (BOSSBS-11997R-CY3)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-A680

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 680)

Artikel-Nr: (BOSSBS-11997R-A680)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyses dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production. Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation. DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues.
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-CY7

Anti-DDAH1 Rabbit Polyclonal Antibody (Cy7®)

Artikel-Nr: (BOSSBS-11997R-CY7)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-CY5

Anti-DDAH1 Rabbit Polyclonal Antibody (Cy5®)

Artikel-Nr: (BOSSBS-11997R-CY5)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R

Anti-DDAH1 Rabbit Polyclonal Antibody

Artikel-Nr: (BOSSBS-11997R)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-A750

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 750)

Artikel-Nr: (BOSSBS-11997R-A750)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyses dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production. Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation. DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues.
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-HRP

Anti-DDAH1 Rabbit Polyclonal Antibody (HRP (Horseradish Peroxidase))

Artikel-Nr: (BOSSBS-11997R-HRP)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-A350

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 350)

Artikel-Nr: (BOSSBS-11997R-A350)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-BOSSBS-11997R-A647

Anti-DDAH1 Rabbit Polyclonal Antibody (Alexa Fluor® 647)

Artikel-Nr: (BOSSBS-11997R-A647)
Lieferant: Bioss
Beschreibung: DDAH, a dimethylarginine dimethylaminohydrolase, hydrolyzes dimethyl arginine (ADMA) and monomethyl arginine (MMA), both inhibitors of nitric oxide synthases, and may be involved in in-vivo modulation of nitric oxide production (1,2). Impairment of DDAH causes ADMA accumulation and a reduction in cGMP generation (3). DDAH II, the predominant DDAH isoform in endothelial cells, facilitates the induction of nitric oxide synthesis by all-trans-Retinoic acid (atRA) (4). DDAH proteins are highly expressed in colon, kidney, stomach and liver tissues (1).
VE: 1 * 100 µl
Image Unavailable-02321-10G-F

2,6-Pyridindicarbonsäure, Supelco®

Lieferant: Merck
Beschreibung: 2,6-Pyridindicarbonsäure, Supelco®

Image Unavailable-8.00614.1000

2,6-Pyridindicarbonsäure zur Synthese, Sigma-Aldrich®

Lieferant: Merck
Beschreibung: 2,6-Pyridindicarbonsäure zur Synthese, Sigma-Aldrich®

Sicherheitsdatenblätter

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Lager für diesen Artikel ist begrenzt, kann aber in einem Lagerhaus in Ihrer Nähe zur Verfügung. Bitte stellen Sie sicher, dass Sie in sind angemeldet auf dieser Seite, so dass verfügbare Bestand angezeigt werden können. Wenn das call noch angezeigt wird und Sie Hilfe benötigen, rufen Sie uns an 1-800-932 - 5000.
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